Delta A

Ministero della salute
Food supplement notified to the MINISTRY OF HEALTH
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29.95

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PACK:
60 capsules in glass bottle.

PRECAUTIONS FOR USE:
This product does not interfere with any medication.
It can even be taken by children over the age of 6, the elderly and pregnant women. We do not recommend taking this supplement only in case of severe liver failure and cirrhosis.

 

MAIN INGREDIENTS:

VITAMIN A 100 % NATURAL

COD LIVER OIL

 

INGREDIENTS:
Cod liver oil (fish); maltodextrins; beta-carotene, coating agent: hydroxy-propyl-methylcellulose (capsule casing), anti-caking agents: magnesium salts of fatty acids.

Last updated on 21/01/2019

 

HOW ITS COMPONENTS WORK:
Modern life often leads to an unbalanced diet.
Industrial agriculture produces food that is poor in nutrients and medicines cause the chronic or acute alteration of the balance of bacterial flora that acts on the absorption of vitamins and minerals.
Alcohol, coffee, tobacco, stress, sugar, the contraceptive pill, antibiotics and anti-cancer chemotherapy are the enemies of vitamins.
Nutritional deficiencies increase with age.
The consequence of all this is the emergence of more or less significant deficiencies in the population as a whole (WHO 1985).
Retinol or vitamin A has extraordinary properties that continue to amaze researchers and chemists around the world.

Its action is proven on:
– visual functions
– skin and mucous membranes
– the synthesis of mucopolysaccharides
– lysosome and mitochondrial membranes (healing)
– reproduction
– cancer prevention and direct action against cancer. THEY ARE PARTICULARLY USEFUL DURING CHEMOTHERAPY TREATMENTS
– boosting cancer therapies
– immunity
– infectious diseases and multiple sclerosis

Vitamin D3 or cholecalciferol is transformed by the body into biologically active calcitriol. It intervenes in the balance of calcium and phosphorus, cell differentiation, immunity and cancer prevention.

WHAT ARE THE SYMPTOMS OF DEFICIENCIES?
– Absence of lacrimation and inflamed eyes
– Decreased night vision
– Dry skin and mucous membranes
– Diarrhoea
– Reduced resistance to infections
– Loss of sense of smell
– Delayed growth

RECOMMENDED USES:

we recommend taking one capsule a day.

 

 

DELTA A can be taken either during or after a meal (the presence of fats favours the absorption of beta-carotene).
It is advisable to combine vitamin A with vitamin E and vitamin C (DELTA E and DELTA C).

It is advisable to take vitamin A on a constant basis to benefit from its preventive action against cancer, as demonstrated by scientific work.

SCIENTIFIC BASES – BIBLIOGRAPHY:
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Vitamin A: ajuvant and steroïd antagonist in the immune response. The journal of Immunology. Vol. III n°5, nov. 1973 : 1376-1380
• BOLLAG W.
Vitamin A and retinoïds : from nutrition to pharmacotherapy in dermatology and oncology. Lancet, 1983 : 860-863.
• BOLLAG W., HANCK A.
From vitamin A to retinoïds, modem trends in the field of oncology and dermatology. Acta vitamin. Enzymal.
1977,31 : 113-125.
• BOUTWELL RK., VERMA AF.
Effects of vitamin A and retinoïds on the biochemical processus linked to carcinogenesis. Pure Appl. Chem. 1979, 51 : 857-866.
• BROC P., STORA C., BERNHEIM L.
De I’emploi de la vitamine A dans le traitement des mastoses. Ann. Endocrin. 1956, 2 : 193-200.
• CHU EW., MALMGREN RA.
An inhibitory effect of vitamin A on the induction of tumors of forestomach an cervix in the Syrian hamster by carci-nogenic polycyclic hydrocarbons. Cancer Res. 1965, 25 : 884-885.
• COHEN SM., WITTENBERG JF., BRYAN GT.
Effects of avitaminosis A and hypervitaminosis A on urinary blodder carcinogenicity of N-(4-(5-Nitro-2. furyl)– 2-Thiazolyl) formide. Cancer Res. 1976, 36 : 2334-2339.
• DAVIES RE.
Effects of vitamin A on 7,12 – dimethylbenz(a) anthracene-induced papillomas in rhino mouse skin. Cancer Res. 1967, 50 : 439-444.
• DENNERT G.
Effects of retinoïc acid on the immune system : stimulation of T killer cell induction. Eur. J. Immunol. 1978, 8, n° 1 : 23-29.
• ELIAS PM., FRIENDS DS.
Vitamin A induced mucous metaplasia : an in vitro system for modulating tight and gap junction differenciation. J. Cell. Biol. 1976, 68 : 173-188.
• HAJJI QMAR
Cancers du sein métastaseés et vit. A. Thése Faculté de Bobigny 1984.
• HOGAN B.
Epithelial cancer, differenciation and vitamin A. Nature. 1979, 277 : 261-262.
• ISRAEL L., AGUILERA J.
Vitamine A et cancer. Path. Biol. 1980, 28, n° 4 : 235-259.
• ISRAEL L., EDELSTEIN R.
Plasmapheresis in patients with disseminated cancer = clinical results and correlation with changtes in serum pro-teins. The concept of non specific blocking factors. Cancer. 1977, 40, 6 : 3145-3146.
• IVAN CAMERON, BARRY GRUBLAS, WAID ROGERS
High-dose methyl prednisolone, vitamin A, and vitamin C in rats bearing the rapidly growing Morris 7777 hepa-toma. Cancer treatment reports, vol. 63, n° 3, march 1979.
• JEROME J., DECOSSE MD.
Potential for chemoprevention. Cancer. 1982, 50 : 2550-2553.
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Relation of vitamins to the sexolands, in ALLEN F., DANFORTH CH., DAISSY EA. (Edss) : sex and internal secretions. Baltimore, Williams et Wilteins CO, 1939.
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Freeze-fracture techniques and applications to the structural analysis of the mammalian plassma membrane,
in Poste G., NICOLSON GL. (Ed) cell surface reviews. Amsterdam North Holland Publishing Co. 1977, vol. 4, pp 75-126.
• MC NUTT NS., WEINSTEIN RS.
Carcinoma of the cervix : deficiency of nexus intercellular junctions. Science 1979, 165 : 597-599.
• MlCKSCHE M., WRBA H.
Stimulation of immune response in lung cancer patients by vitamin A therapy. Oncology, 1977, 34 : 234-238.
• MOON RC., GRUBBS CJ., SPORN MB.
Retinyl acetate inhibits mammary carcinogenesis induced by N-methyl-N-nitrourea. Nature. 1977, 267 : 620- 621
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The influence of vitamin A on the susceptibility of the rat lung to 3-methyl cholantrone. Int. J. Cancer. 1976, 17 : 351–387.
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Induction fo ornithine decarboxylase as an early possibly obligatory event in mouse skin carcinogenesis.
Cancer Res. 1976, 36, 2644.
• O’BRIEN TG., SIMSIMAN RC., BOUTWELL RK.
Induction of polyamine-synthetic enzymes in mouse epidermis and their specificity for tumor promotion.
Cancer Res. 1975, 35 : 2426-2433.
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Mucous metaplasia and gap junctions in the vitamin A acid treated skin tumor kerato acanthoma. Cancer Res. 1975, 35 : 364-369.
• RUSSEL DH., DURIE GM.
Polyamines as biochimical Markers of normal and malignant growth. New York, Rwen Press, 1978.
• SAFFIOTTI U., MONTESANO R., SELLAKUMAN AR.
Experimental cancer of the lung: inhibition by vitamin A of the induction of tracheobronchial squamous metaplasia and squamous cell tumors. Cancer. 1967, 20 : 857-864.
• SPORN MB., DUNLOP NM., NEWTON DL.
Prevention of chemical carcinogenesis by vitamin A and its synthetic analogs. Fed. Proc. 1976, 35 : 1332- 1338.
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Chemoprevention of cancer with retinoïds. Fed. Proc. 1979, 38 : 2528-2534.
• STEPP W.
Versuche über Futterung mit Lipoid frever Nahrurg. Biochem. Z. 1909, 22 : 452-460.
• TODARO GJ., DE LARCO E., SPORN MB.
Retinoïds block phenotypic cell transformation produced by sarcoma growth factor. Nature. 1978, 276 : 272- 274.
• TODARO GJ., HUEBNER RJ.
The vival oncogene hypothesis: New evidence. Proc. Nat. Acad. Sci. USA. 1972, 69 : 1009-1115.
• VENANZIE F., MONTENEGRO O.
Arachives brasil Med, 1947, 37 : 135-147 et Revista de obst. et Gyn. Brasil, 1947, 8 (n° 2) : 70-89.
• WEINSTEIN RS., MERK FB., ALROY J.
The structure and function of intercellular junctions in cancer. Adv. cancer. Res. 1976, 23 : 23-89.
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Tissue changes following deprivation of fat-soluble A vitamin. J. Exp. Med. 42 : 753-778.
• WOLBACH SB., HOWE PR.
Vitamin A deficiency in guinera-pig. Arch. Pathol. Lab. Med. 1928, 5 : 239-2563.
• BRANDES T., SATO S., VEDA H.
Radiation response of L1210 Leukemia cells pretreated with vitamin A ahohol.J. Nat. Cancer. Invest. 1974, 52 : 945–949.
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Enhancement of the antitumor effect of 1,3 bis 2 chloroethyl 1 nitrosourea and cyclophosphamide by vitamin A. J. Nat. Cancer. Inst. 1972, 48 : 921-926.
• NAKASHIMA T., NAGAI T., MAKISHIMA K., HIROTO I.
Primary culture of carcinoma of the maxillary sinus in human. Jibiinkoka Rinsho. 1978, 1 : 78-83.
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Fondamenti di Nutrizione Umana. Il Pensiero Scientitico Editore.
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Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3. FASEB J. 2005 Jul.19(9):1067-77.
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1,25-dihydroxyvitamin D3 is a direct inducer of antimicrobial peptide gene expression. J Immunol. 2004 Sep 1;173(5):2909-12. Erratum in: J Immunol. 2004 Nov 15;173(10):following 6489. Hanrahan. JH [corrected to Hanrahan, JW]. (http://www.ncbi.nlm.nih.gov/entrez/query. fcgi? db=pubmed&cmd=
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Weight 0.132 kg